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El. knyga: Mechanisms in B-Cell Neoplasia 1988: Workshop at the National Cancer Institute, National Institutes of Health, Bethesda, MD, USA, March 23-25, 1988

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Mechanisms in B-Cell Neoplasia 1988: Workshop at the National Cancer Institute, National Institutes of Health, Bethesda, MD, USA, March 23-25, 1988Didesnis paveikslėlis
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The papers in this book were presented at the 6th Workshop on Mechanisms in B-Cell Neoplasia, held in Bethesda, March 23-25, 1988. On alternate years this meeting is sponsored by the . ;. Basel Institute of Immunology in Basel, Switzerland and by the National Cancer Institute in Bethesda, and is attended by 100 to 150 parti­ cipants. This 6th workshop, like the preceding five, was characterized by intense and enthusiastic discussion which reflects, we think, the exciting growth and development of this field. It is quite clear, however, that despite many general advances an understanding of the precise underlying mechanisms in B-cell tumor development is not yet defined. Probably, there is no single mechanism for all the various forms of B-cell neo­ plastic development. Many different forms of B-cell neoplasms are known, and these are distinguished by several characteristics: 1) the stage of development attained by the tumor stem cells; 2) mode of growth (slow or fast); 3) association with natural or inductive etiologic agents and 4) specific and consistent mutational mechanisms such as retroviral insertion, chromosomal rearrangement. Those charac­ teristic forms which arise naturally in relatively high frequency or those tumors with hallmark properties which can be induced consistently are the models most frequently studied, e. g. , endemic Burkitt's lymphoma, follicular lymphoma, acute and chronic lymphocytic leukemia and mUltiple myeloma in man; bursal lymphoma in chickens; Abelson virus induced pre B cell lymphomas and plasmacytomas in mice and immunocytomas in rats. Each model system, has special problems and advantages.

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Springer Book Archives
I: Pathogenetic Mechanisms.- Early B Cell Tumors.- B Cell Precursors in
Bone Marrow: In Vivo Proliferation, Localization, Stimulation by Activated
Macrophages and Implications for Oncogenesis.- Lymphoid-Restricted Stem
Cells.- Recombinant Interleukin-7 Supports the Growth of Normal B Lymphocyte
Precursors.- Mac-1+ Bone Marrow Cells Include Precursors of B Cells and T
Cells.- Molecular Characterization of a Transforming Retrovirus Involved in
Pre-B Cell Lymphomas.- In Vitro Transformation of Murine Bone Marrow Cells
with a v-raf/v-myc Retrovirus Yields Clonally Related Mature B Cells and
Macrophages.- Closely Related BCR/ABL Oncogenes Are Associated with the
Distinctive Clinical Biologies of Philadelphia Chromosome Positive Chronic
Myelogenous and Acute Lymphocytic Leukemia.- The Pathogenesis of Tumors
Induced by Helper Virus-Free Abelson Murine Leukemia Virus.- The Development
of Three Distinct Avian B Cell Lymphomas.- Bursal Stem Cells as Targets for
myc-Induced Preneoplastic Proliferation and Maturation Arrest.- Molecular
Analysis, of an AIDS-Associated Burkitts Lymphoma: Near-Identity with
Endemic Cases.- B Cell and Plasma Cell Tumors.- Transgenic Mouse Models for
Hematopoietic Tumorigenesis.- Genetic Studies on Eµ-myc Transgenic Mice.-
Differential Expression of myc-family Genes During Development: Normal and
Deregulated N-myc Expression in Transgenic Mice.- Plasmacytoma Induction by J
Series of v-myc Recombinant Retroviruses: Evidence for the Requirement of Two
(raf and myc) Oncogenes for Transformation.- Synergy of an IgH
Promoter-Enhancer-Driven c-myc/v-Ha-ras Retrovirus and Pristane in the
Induction of Murine Plasmacytomas.- The Genetics of Susceptibility to
RIM-Induced Plasmacytomagenesis.- Significance of Chromosome 8 Breakpoint
Location in BurkittsLymphoma: Correlation with Geographical Origin and
Association with Epstein-Barr Virus.- B Cell Proliferation in Follicles,
Germinal Centre Formation and the Site of Neoplastic Transformation in
Burkitts Lymphoma.- Altered Growth Phenotype of a Burkitts Lymphoma Line
Following the Introduction and Stable Expression of the EBNA 2A Gene.-
Soluble CD23/BLAST-2 (S-CD23/Blast-2) and Its Role in B Cell Proliferation.-
Burkitts Lymphoma Variant Translocations: Distribution of Chromosomal
Breakpoints and Perturbated Regulation of a Mutated c-myc Gene.-
Differentiation Associated c-myc Expression in Phorbol Ester and Lymphokine
Stimulated B-Type Chronic Lymphocytic Leukemia Cells.- B and T Cell Responses
Induced by Interleukin-6.- II: Studies of B Cell Relevant Oncogenes.- c-myc.-
Prediction of a Dimerization Surface Common to a New Class of
Sequence-Specific DNA Binding Proteins.- Immunological Probes in the Analysis
of myc Protein Expression.- The Replication Activity of SV40 DNA Correlates
with the Level of c-myc Expression in Human Tumor Cell Lines.- Elevated
Levels of the c-myc Protein in Blooms Syndrome and Induction of c-myc by DNA
Strand Breakage.- Deregulation of the c-myc and N-myc Genes in Transformed
Cells.- Stimulation of the Antigen Receptor Complex Leads to Transcriptional
Activation of the c-myc Gene in Normal Human T Cells.- A Repressor of c-myc
Transcription Is Found Specifically in Plasmacytomas.- Regulation of c-myc
Gene Transcription in B Lymphocytes: Mechanisms of Negative and Positive
Control.- Multifactorial Regulation of the Human c-myc Oncogene.- Negative
Control Elements Within and Near the Murine c-myc Gene.- Disruption of the
Putative c-myc Auto-Regulation Mechanism in a Human B Cell Line.- Deregulated
Expression of an Activated Alleleof Human c-myc in Transfected Fibroblast
Cultures.- Comparison of c-, N-, and L-myc Transcriptional Regulation.-
Delivery of c-myc Antisense Phosphorothioate Oligodeoxynucleotides to
Hematopoietic Cells in Culture by Liposome Fusion: Specific Reduction in
c-myc Protein Expression Correlates with Inhibition of Cell Growth and DNA
Synthesis.- Different Biological Effects of c-myc and H-ras Oncogene
Expression in EBV-Infected Human Lymphoblasts.- c-abl.- Abrogation of IL-3
Dependence of Myeloid FDC-P1 Cells by Tyrosine Kinase Oncogenes Is Associated
with Induction of c-myc.- U.R. RAPP The Abelson Protein Is Required for
Initiation and Maintenance for Transformation in Murine Pre-B Cells.- c-myb.-
Expression and Function of the c-myb Oncogene During Hematopoietic
Differentiation.- Sense and Anti-sense Transcription in the Murine c-myb
Attenuator Region.- bcl-2.- Expression of the Murine Proto-Oncogene bcl-2 Is
Stage Specific and Cell-Type Specific.- Recent Progress on the Human bcl-2
Gene Involved in Follicular Lymphoma: Cheracterization of the Protein
Products.
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